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The Journal of Thoracic and Cardiovascular Surgery, Vol 92, 402-405, Copyright © 1986 by The American Association for Thoracic Surgery and The Western Thoracic Surgical Association
SY DeLeon, MN Ilbawi, FS Idriss, AJ Muster, SS Gidding, TE Berry and MH Paul
Four of 44 patients who had undergone the Fontan operation had persistent
low cardiac output necessitating takedown of the shunt 6 to 65 hours
(average 23 hours) postoperatively. All four were in a group of 22 patients
with complex lesions other than tricuspid atresia with ventriculoarterial
concordance. The development of postoperative right atrial hypertension
(average 24 torr), hepatomegaly, marked ascites, and decreasing lung
compliance led to severe systemic hypotension with systolic arterial
pressure ranging from 55 to 82 torr (average 68 torr), persistent metabolic
acidosis, and oliguria despite massive colloid and crystalloid infusions
(11,000 ml/m2/24 hr) and inotropic support. At reoperation the
atriopulmonary anastomosis, which was found to be wide open, was taken down
and an atrial septal defect was created in all patients. Three patients
were left with a Glenn shunt and an aortopulmonary shunt to the left lung.
One patient had bilateral aortopulmonary shunts. Two patients who survived
reoperation had immediate postoperative improvement in systolic arterial
and mean right atrial pressure (average 100 torr and 11.5 torr,
respectively). Both are well 5 months and 4 years later. Repeat Fontan
operation remains a possibility with acceptable risks because of the
presence of the Glenn shunt in both patients. We believe that takedown
should be considered in patients with persistent low cardiac output after
the Fontan operation.
ARTICLES
Persistent low cardiac output after the Fontan operation. Should takedown be considered?
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