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J Thorac Cardiovasc Surg 2004;128:17-20
© 2004 The American Association for Thoracic Surgery
Editorial |
a Centre Cardio-Thoracique de Monaco, Monte Carlo, Monaco
Received for publication February 3, 2004; accepted for publication February 25, 2004.
* Address for reprints: Vincent Dor, MD, Centre Cardio-Thoracique de Monaco, 11 bis, Avenue d'Ostende, Cedex Monte Carlo MC 98004, Monaco
vdor@ccm.mc
| The first 300 words of the full text of this article appear below. |
The Cleveland Clinic group's article in this issue, entitled "Neurohormonal Response to Left Ventricular Reconstruction Surgery in Ischemic Cardiomyopathy" and written by Schenk and collaborators,1 is excellent. It should be read by cardiac surgeons and I hope by cardiologists, because its synthesis is a well-based confirmation that ischemic dilated failing ventricles can be improved by endoventricular circular reorganization. What a long way we've come since 1984, when all presentations of this technique to surgical and medical audiences were followed only by skeptical questions.
I appreciate the use by Schenk and collaborators1 of the term "left ventricular reconstruction" (LVR), which avoids the confusion contained in the inappropriate "surgical remodeling" and is equivalent to the other names given later to the same procedure, such as "endoaneurysmorrhaphy" or "surgical ventricular restoration." The last word has, for the French, a poor political connotation.
This technique has been used since 1984, and the results of this serious, complete, precise, didactic article, coming from a renowned institution, allow us to assess whether the reality is in concordance with the aim. This technique was established to optimize the surgical procedure of extensive blind endocardiectomy proposed by Josephson and colleagues2 to treat intractable recurrent ventricular tachycardias before the implantable defibrillator era. The goal was to improve the results, which were excellent for reducing lethal arrhythmias but extremely poor for augmenting left ventricular performance.
The aim of LVR was to use a circular patch sutured inside the ventricle on contractile myocardium to exclude all nonresectable asynergic areas (primarily septal), to reorganize the curvature of the distended wall, and to do all this without excessive volume reduction of the left ventricular cavity. I fully agree with four important points focused on by Schenk and collaborators1; however, another point is questionable and yet another must be discussed.
Point one
The 15 patients
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